The system's response to this Zika virus, instead of the virus may be liable according to a Yale study. This insight could lead to new means of treating patients the researchers stated.
Infection with Zika virus causes death and paralysis, in mice versions lacking an antiviral response. To understand the mechanics, a research team led by immunobiologist Akiko Iwasaki examined the spread of disease.
The study team discovered that when the Zika disease spreads from the circulating blood to the brain, immune cells called CD8 T cells flooding the brain. Zika-related paralysis is also triggered by them while these T cells sharply limit the disease of nerve cells, the researchers stated.
"The immune cells that are generated by infection start attacking our very own neurons," Iwasaki said. "The damage isn't occurring through the virus disease, but instead the immune reaction to the virus."
Immune-mediated nerve damage underlies syndrome, which affects many individuals infected with the Zika virus. The research findings indicate that suppressing the immune response might be an approach to treating the syndrome, which causes weakness and, in rare cases, paralysis.
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